March 4, 2020

Du, X., et al. (2020) Loss-of-function BK channel mutation causes impaired mitochondria and progressive cerebellar ataxia. PNAS.

March 25, 2019

Du, X., et al. (2019) α1ACT is essential for survival and early cerebellar programming in a critical neonatal window. Neuron. Online Publication

February 10, 2018

Du, X., et al. (2018) Spinocerebellar Ataxia Type 6: Molecular Mechanisms and Calcium Channel Genetics Adv Exp Med Biol. 2018;1049:147-17

January 26, 2018

Hejazi Pastor, DP., et al. (2018) Targeting the CACNA1A IRES as a Treatment for Spinocerebellar Ataxia Type 6. Cerebellum. 2018 Feb;17(1):72-77

July 13, 2016

Miyazaki, Y., et al. (2016) An miRNA-mediated therapy for SCA6 blocks IRES-driven translation of the CACNA1A second cistronSci Transl Med 8, 347ra394

May 7, 2015

Tsou, WL., et al. (2015) DnaJ-1 and karyopherin α3 suppress degeneration in a new Drosophila model of Spinocerebellar Ataxia Type 6Hum Mol Genet. 24(15):4385-96

July 3, 2013

Du, X., et al. (2013) A second cistron in the CACNA1A gene encodes a transcription factor that mediates cerebellar development and SCA6Cell 154, 118-133


February 1, 2017

Karginov, TA., et al. (2017) Mammalian Polycistronic mRNAs and DiseaseTrends in Genetics. 33(2):129–142

March 15, 2014

Du, X., et al. (2014) Revelations from a bicistronic calcium channel geneCell Cycle 13, 875-876



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